Overall, oxidative burst can induce higher activity of the antioxidative system which is governed by enzymatic activity, such as superoxide dismutase (SOD), catalase (CAT), and peroxidase (POX), as well as defense-related enzymes chitinase (CHI) and phenylalanine ammonia-lyase (PAL), this one leading to an early (<18 h after infection (hai)) and more intense fluorescence of epidermal cells due to the accumulation of phenylpropanoid derivatives at the infection site [3,4]. This evidence concerns the gene SOD1 and infection.