In addition, it is known that dysregulated mRNA translation, specifically the irregular mRNA circularization driven by PABPC1‐eIF4F complex, acts a pivotal role in the intricate chain of events that precipitate cancer onset.[19, 21, 22] Translation serves as a foundational mechanism exploited by cancer cells to sustain their malignant characteristics[18] Our results further elucidated the preferential non‐m6A translation of specific mRNAs regulated by hnRNPA2B1‐PABPC1 axis. Here, HNRNPA2B1 is linked to cancer.