These data are consistent with the findings showing the highest expression of FABP4 in skin TRM cells among native T cells and various memory T cell types[13] and FABP4 promotes survival of skin TRM cells[13] and cancer cells in gastric adenocarcinoma[17] and colon cancer.[31] Congruent with the high circulating fatty acid levels in T1D patients resulting from defective insulin‐mediated suppression of lipolysis,[32] this finding reinforces the pathogenic significance of fatty acids in prolonging TRM cell‐mediated pancreatic autoimmune attack on top of their toxicity on β cell apoptosis. The gene discussed is FABP4; the disease is type 1 diabetes mellitus.