Since CD8+ T cells are recruited into infected tissues mainly triggered by CXC family chemokines including C‐X‐C motif chemokine (CXCL) −9 and −10 stimulation on their surface C‐X‐C motif chemokine receptor 3 (CXCR3),[23] and CXCL10 was originally identified as an IFNγ inducible protein in T1D,[24] we next investigated whether FABP4 promotes recruitment of CD8+ T cells into pancreatic islets via enhancing the secretion of CXCL10 from TRM cells. Here, FABP4 is linked to type 1 diabetes mellitus.