Diazepam has a protective effect on the development of experimental autoimmune encephalomyelitis by reducing the incidence of the disease by reducing the number of inflammatory cells in the central nervous system; furthermore, diazepam can reduce the levels of the biomarker transposon protein translocator protein (TSPO), which is a biomarker of inflammatory diseases in brain tissue and inflammatory CD11b+ cells in the central nervous system [12]. This evidence concerns the gene TSPO and experimental autoimmune encephalomyelitis.