Significant studies have shown miR-98’s diagnostic precision in differentiating PCa patients from healthy subjects and its association with key pathways and oncogenes, such as the oncogene HMGA2 via the lncRNA NEAT1. Additionally, the potential modulation of miR-98 by factors like vitamin D and dietary tomato carotenoids emphasizes its multifaceted role in prostate cancer development, progression, and potential therapeutic interventions. Here, NEAT1 is linked to posterior cortical atrophy.