CALB2 and schizophrenia: Although neuronal calretinin expression is reported to be unchanged in the prefrontal cortex in schizophrenia,16,60–62 the density of calretinin-immunopositive neurons has been found to be reduced in the caudate nucleus in the disorder.63 We also found enrichment of schizophrenia common variant liability in genes with high expression specificity for 3 populations of developing GABAergic MSN populations destined for the striatum.