GAPDH and fetal growth restriction: As widely acknowledged, the resilient and immune‐rich environment of the liver supports regional and systemic homeostasis, enabling hepatic regeneration.[51] However, the delicate equilibrium of liver homeostasis is perturbed by the process of intrauterine programming caused by an unfavorable intrauterine environment.[52] It is well‐established that IUGR leads to intrauterine hypoxia, a fact validated by data showing an enrichment of the HIF‐1 signaling pathway and highlighting the expression of EPO and GAPDH during IUGR.