For example, claudin-4 is associated with developing therapy resistance independently (2), as are LAT1 (62) and SLC1A5.(63) Additionally, a key phenotype during ovarian cancer development is ascites formation, which is a hallmark of ovarian cancer malignancy and is closely related to the availability of metabolites for tumor cells (64–67), which can also negatively modulate immune cells (68). This evidence concerns the gene SLC7A5 and neoplasm.