SQSTM1 and dengue disease: Our data underscores that dengue virus institutes a block in autophagy flux in primary DCs, as evident by accumulation of LC3-II and p62 in the presence of dengue virus (Figures 3A–E) but a lack of LC3-II accumulation upon concomitant dengue virus infection and bafilomycin treatment (Supplementary Figures 2A, B).