Liu et al. established a cellular myocardial infarction (MI) model after administration of 20 μg/mL and found that the inhibitory effect of oxygen-glucose deprivation/reperfusion (OGD/R) on cell viability was reversed and the levels of MDA, ROS, and Fe2+ were downregulated, and the expression of GSH, SLC7A11, and GPX4 was increased. Here, SLC7A11 is linked to myocardial infarction.