Abdominal obesity is associated with the development of enlarged and dysfunctional adipose cells (21), which secrete adipokines like adiponectin and inflammatory cytokines, including interleukins and tumor necrosis factor alpha, which contribute to insulin resistance as well as the pro-inflammatory and pro-hypertensive states associated with visceral obesity (22–24). The gene discussed is ADIPOQ; the disease is Abdominal obesity.