However, the prevailing theory is that EBV predisposes the host to MS-related autoimmunity through the infection of host B cells as well as through molecular mimicry of host myelin-associated proteins myelin basic protein (MBP) and glial cell adhesion molecule (GlialCAM) by the EBV nuclear antigen 1 (EBNA1) [105, 106, 159]. This evidence concerns the gene MBP and Autoimmunity.