The pathogenesis of LVA secondary to viral myocarditis is unclear (13), which may be in connection with the long-term fiber scar repair of the necrotic myocardium (4), while Th17 cells/IL-17A-mediated autoimmunity and myocardial fibrosis may involve DCM formation post-myocarditis (40). This evidence concerns the gene IL17A and viral myocarditis.