The relation found between CXCL9 plasma levels, a chemokine implicated in Th1 response, CD8 T cells, and NK cell trafficking (28) and recently reported for its implication in the pathogenesis of pediatric cerebral malaria (18), and LILRB2 revealed opposite immune mechanisms between D3 and D30. The gene discussed is CXCL9; the disease is cerebral malaria.