Indeed, compared to HC and IPF patients (which again overall behaved similarly), M-COV1/2 PBMCs secreted, upon infection with live PAO1, less IL-8, MIP-1α, IL-1α, IL-1b, TNF-α, G-CSF, MIP-2α, IL-23 and IL-10 (Figures 7A–I). This evidence concerns the gene CCL3 and idiopathic pulmonary fibrosis.