Multiple studies have shown that Th17 cells are considered an important group of cells that mediate inflammatory responses, and the release of interleukin-17A (IL-17A) by Th17 cells contributes to the maintenance of chronic inflammatory states and the development of a cancer-promoting microenvironment, which is closely related to the occurrence and development of PCa [10–13]. The gene discussed is IL17A; the disease is posterior cortical atrophy.