Case reports also include limbic encephalitis with N-methyl-D-aspartate receptor (NMDAR) autoantibodies [11] or autoantibodies against glutamic acid decarboxylase 65-kilodalton isoform (GAD65) [12], cerebellar hypoplasia [13], demyelinating polyneuropathy [14], autoimmune cerebellar degeneration with ataxia and a preceding Miller-Fisher syndrome [15], epilepsy and limb girdle muscular dystrophy [16], and myopathy [17]. The gene discussed is GAD2; the disease is cerebellar ataxia.