SLC2A1 and glioblastoma: Chédeville et al. [58] revealed that hypoxia-induced upregulation of LDHA in glioblastoma triggers a glycolytic shift accompanied by enhanced expression of pro-angiogenic factors, including VEGFA, with high expression levels of genes, such as SLC2A1, LDHA, PDK1, PFKFB4, HK2, VEGFA, SERPINE1, TFRC, and ADM significantly associated with decreased survival rates.