BCL9 and neoplasm: The expression of phospho-TAK1, phospho-NF-κB p65, and IRF1 of intratumoral cDC1 were upregulated from hsBCL9z96-treated CT26 tumor-bearing mice, as well as from MC38 tumor-bearing Bcl9/Bcl9l deficiency mice (Supplementary Fig. 10h–m).