Moreover, fibroblast‐specific deficiency of Mettl1 has been shown to attenuate cardiac fibroblast activation and fibrosis following myocardial infarction.[17] However, the role of Mettl1/m7G in non‐ischemic cardiac hypertrophy and HF remains unexplored. Here, METTL1 is linked to myocardial infarction.