The identification of various cell subclusters co-expressing lncRNA-SNHG14, MRTFA, and MRTFB is consistent with the observation that MKL1/MRTFA is abnormally aggregated in human AD postmortem brain tissues [58], indicating that the disruption of MKL1/MRTFA function may contribute to the progression of AD pathology [59]. This evidence concerns the gene MRTFA and Alzheimer disease.