SMAD2 and neuroblastoma: Whereas all upregulated in NB co-cultured with TME cells, MCL-1, BCL2 and survivin were not upregulated in TGF-β1-treated NB cells and their expression were not affected upon pharmacological or genetic inhibition of the TGF-β1/TAK1/NF-κB/SMAD2 signalling pathway, suggesting a different mechanism of regulation.