This family extends the clinical spectrum of GLUT1‐DS, ranging from milder classic phenotypes to more subtle neurological disorder carriers, further strengthening the idea that familial GLUT1‐DS is less severe than the sporadic form [2, 3, 4, 5, 6, 7, 8, 9, 10, 11]. This evidence concerns the gene SLC2A1 and Dravet syndrome.