Further, ‘thyroid hormone signalling pathway’, ‘Notch signalling pathway’, and ‘antiviral mechanism by IFN-stimulated genes’ were significantly enriched for genes overlapping T2D and IBS (Supplementary Data 53), while ‘primary hypercortisolism’ (having a potential implication in immune or inflammatory processes) were enriched for genes overlapping T2D and gastritis-duodenitis (Supplementary Data 54). This evidence concerns the gene IFNA1 and duodenitis.