These gene expression levels were, respectively, reduced to 12% and 11%, 35% and 30%, 26% and 20%, and 31% and 27% of those observed with CDDP treatment alone, indicating that HGFK1 could significantly inhibit the activation of the CDDP-induced Wnt/β-catenin and Notch signaling pathways, thus inhibiting the stemness and CDDP-enriched ability of CD90+ CSCs in HCC. Here, THY1 is linked to hepatocellular carcinoma.