One such mechanism is the inhibition of cell proliferation and viability through the modulation of cell cycle regulators (for example, by inducing cell cycle arrest at the G1 phase), while another mechanism involves the induction of apoptosis by the modulation of apoptotic proteins (Bax and Bcl2) or activation of apoptotic signaling pathways (AXL phosphorylation, Akt/mTOR, PI3/AKT, and p-JNK) in lung cancer cells, which are crucial for cancer cell survival and proliferation [78,79,80]. This evidence concerns the gene AKT1 and lung cancer.