Interestingly, and consistent with our current observation of this miRNA’s strong responsiveness to oxidative stress, Harraz et al. showed its neuroprotective function in response to neuronal injury, for example in stroke, transient global ischemia, and neurodegenerative disorders, by regulating the expression of two glutamate receptor genes, NR2B (GRIN2B) and GLUR2 (GRIA2) [45]. This evidence concerns the gene GRIA2 and Stroke.