Recently, Wang et al. established that the expression of SLFN5 was increased in the human lung cell line A549 and a mouse lung pneumonia model by the glycolipid lipopolysaccharide (LPS), an endotoxin produced by Gram-negative bacteria, and the cause for the inflammatory response seen in lungs affected by pneumonia [23]. This evidence concerns the gene SLFN5 and susceptibility to pneumonia measurement.