mTOR signaling is often overactive in cancers and the different isoforms act as key cancer regulators [12]—mTORC1 regulates protein synthesis and autophagy, mTORC2 regulates kinases of the AGC family, and mTORC3 causes chemotherapy resistance, though its specific role in breast cancer is yet to be understood [13]. This evidence concerns the gene MTOR and breast carcinoma.