Molecules such as ICAM-1, VCAM-1, and P-selectin can accentuate the adhesion of monocytes and lymphocytes and the high content of oxidized LDL, lysophosphatidylcholine, and oxidized fatty acids in an active state of dyslipidemia induce the expression not only of these adhesion molecules but also that of sequestering receptors, such as CD-36, SR-A, and LOX-1 [60]. Here, OLR1 is linked to metabolic syndrome.