Furthermore, mice with a deletion of the kinesin family member 3A (Kif3a) in renal tubular epithelial cells develop kidney cysts and show a reduction in tetanic-specific force, a decrease in skeletal muscle mass, a higher percentage of smaller myofibers, a reduction in protein synthesis, and an elevation in atrogenes [107], suggesting that Kif3a−/− mice could be a valuable genetic mouse model to study CKD-associated sarcopenia. The gene discussed is KIF3A; the disease is chronic kidney disease.