LNCARSR and renal carcinoma: For instance, lncARSR, which showed high expression levels in sunitinib-resistant renal cancer cells, has been found to be essential for the resistant phenotype, through competition with endogenous RNA for miR-34 and miR-449, leading to the upregulation of AXL/c-MET and the activation of STAT3, AKT, and ERK pathways.