In the same study, CXCR2+ MPO+ (Myeloperoxidase) cells infiltrated spontaneous intestinal tumors in Apcmin/+ and AhCreER; Apcfl/+; Ptenfl/fl mice and CXCR1/2 blocking pepducin treatment suppressed tumor infiltration of MPO+ cells and adenoma formation concurrently. This evidence concerns the gene CXCR2 and neoplasm.