Moreover, considering the role of some galectins (Gal-3, Gal-9, Gal-10) on both Th1 and Th2 inflammation, it would be interesting to investigate whether these proteins could reliably be used in the presence of asthma and Th1-driven comorbidities such as respiratory infections or bronchiectasis [98]. The gene discussed is CLC; the disease is respiratory tract infectious disorder.