Furthermore, KEGG pathway analysis showed that transcriptionally activated genes, which were GATA1-unique peak genes, were enriched in pathways associated with Maturity onset diabetes of the young (Benjamini p = 2.99 × 10−4), Leukocyte transendothelial migration (Benjamini p = 2.98 × 10−2), and ECM-receptor interaction (Benjamini p = 1.25 × 10−2; Figure 1C). This evidence concerns the gene GATA1 and diabetes mellitus.