It is also presumed that the pathophysiology of oropharyngeal dysphagia in COVID-19 patients is linked to interaction of the virus with angiotensin-converting enzyme 2 (ACE2) and transmembrane serine protease 2 (TMPRSS2) [20], proteins present in anatomical regions specific to the swallowing function, such as the oral, pharyngeal and nasal mucosa. This evidence concerns the gene ACE2 and COVID-19.