Dissection of the biological and disease-associated functions of the IFN-MPs suggested increased blood cell activation, recruitment, movement, adhesion, migration, engulfment, and atherosclerosis in stable CAD, decreased lipid droplet accumulation, blood platelet aggregation, blood cell accumulation, and cell movement in acute MI, and increased infection, cell movement, cell migration, proliferation, engulfment, angiogenesis, fibrogenesis, and vascular development in remote MI (Data S1). This evidence concerns the gene IFNA1 and myocardial infarction.