Although evidence suggests that high levels of HIF1α during chronic hypoxia may lead to glomerular disease and proteinuria (Varga et al., 2018), this finding may be attributed to podocytes being more susceptible to damage during chronic hypoxia, including epithelial‐to‐mesenchymal transition, slit diaphragm dysfunction and disturbances in the cell cytoskeleton caused by HIF accumulation (Dong et al., 2021). The gene discussed is HIF1A; the disease is glomerular disorder.