CALR and Alzheimer disease: Notably, considering the DEPs determined from all 118 cells (for AD vs WT hiPSC‐neurons), gene ontology (GO) profiler intersection analysis (Table S3, Supporting Information) showed several of the altered proteins were involved in related pathways, for example, the highest‐ranking pathway of APOE, CALR, PTPA, AP2M1, SNX6, VGF, and BDNF.