In fact, it appears able to inhibit the EMT process by regulating HIF1α expression [6], or through transcriptional repression of SNAIL1, ZEB1 and VEGF in colon, oral and neck tumours [7, 8], and to repress EMT in pancreatic ductal adenocarcinoma cells sensitizing cancer cells to chemotherapy [9]. Here, SNAI1 is linked to cancer.