JUN and infection: The c-Jun N-terminal kinase (JNK) and P38 kinases, not the extracellular regulated kinases (ERK), of mitogen-activated protein kinases (MAPKs) pathway, a pro-inflammatory pathway activated via bacterial phagocytosis, were markedly inhibited in RAW264.7 with ΔPA0034 infection compared to PAO1 group (Fig. S2B).