This is an interesting finding as tissue‐resident NK cells in adipose tissue of mice fed high‐fat diet can induce insulin resistance by promoting the differentiation of macrophages towards a pro‐inflammatory phenotype through IFN‐γ.[39] Although the overall levels of IFN‐γ and TNF‐α were lower in VAT of DHT‐exposed mice, we did not specifically determine the ability of NK cells to release IFN‐γ. The gene discussed is IFNG; the disease is Insulin resistance.