INS and obesity due to melanocortin 4 receptor deficiency: Contrary, macrophages with an anti‐inflammatory phenotype are essential for maintaining insulin sensitivity in mice with diet‐induced obesity.[7] Eosinophils have been shown to sustain the anti‐inflammatory phenotype of macrophages through the release of IL‐4 and IL‐13.[8] Here we observed a lower frequency of eosinophils as well as reduced levels of IL‐4 and IL‐13 in VAT of DHT‐exposed mice, which could potentially influence the function of macrophages in adipose tissue.