Alterations in the drivers commonly affected in the other brain tumours, including IDH1/2, TP53, ATRX, TERT, EGFR and QKI, were not detected.1 Across the LGNT samples, mutual exclusivity was observed between the recurrent genomic alterations in BRAF, FGFR1, FGFR3 and PDGFRA (Fig. 3A). Here, QKI is linked to brain neoplasm.