Interestingly, a previous study revealed that APS alleviated inflammatory damage of the BPD cell model through inhibiting the activation of nuclear factor-κB (NF-κB) and reducing mRNA and protein expression levels of IL-8 and intercellular adhesion molecule 1 (ICAM-1) in bronchopulmonary dysplasia (BPD) (110). Here, CXCL8 is linked to bronchopulmonary dysplasia.