Knocking down SLC27A2 increased the proportion of apoptotic cells in BC, and providing long-chain fatty acids (FAs) under cellular starvation conditions could rescue starvation-induced cell death but could not rescue the death of SLC27A2 knocked-down cell lines, indicating that knocking down SLC27A2 may lead to disturbances in cellular lipid uptake and transport (Fig. 8d). The gene discussed is SLC27A2; the disease is breast cancer.