To investigate the cross-interactions of quercetin, quercetin-A1a, and quercetin-A1a1 with other AD-related targets, we performed similar molecular docking of these three compounds into the structures of DAPK1, CDK1, PLK1, IGF1R, c-MET, EGFR, DRD4. This evidence concerns the gene PLK1 and Alzheimer disease.