Two overlapped upregulated genes by HPV16-miR-H1 and HPV16-miR-H6 have been proven to play important roles in cervical cancer: FGFR2 inhibited cervical cancer cell viability and invasion [33], CCDC7 activated interleukin-6 and vascular endothelial growth factor to promote proliferation via the JAK-STAT3 pathway in cervical cancer cells [34]. This evidence concerns the gene FGFR2 and cervical cancer.