CXCL10 and infection: Indeed, VSVΔ51 infection induced a large cross-section of antiviral genes (IFITs, IFITMs, OASs, ISGs) and different pro-inflammatory cytokines and chemokines (CCL5, CXCL10, IFNB1) that were all downregulated by 4-OI treatment (Fig. 5a, b and Fig. S7b).