Persistent injury leads to proteinuria and a progressive decline in GFR.1, –3 FSGS is classified into primary, genetic, secondary, and undetermined cause, differing in prognosis and management.4 Primary FSGS clinically manifests through nephrotic syndrome and is marked by substantial proteinuria, low serum albumin levels, hypertension, elevated cholesterol, and edema. This evidence concerns the gene ALB and nephrotic syndrome.