Performing the same analysis on merged WT1/AR peaks, we found 59 were co-located with H3K4me3 peaks (63.2%, P-value < 0.001 one-sided Fishers exact test), (Figure 5A, Supplementary Table S5), further substantiating that any competitive binding between WT1 and AR in PCOS patients is likely to result in aberrant regulation of genes that are actively transcribed during decidualization. The gene discussed is WT1; the disease is polycystic ovary syndrome.